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Clenbuterol belongs to a class of drugs called beta-2 agonists, which work by stimulating beta-2 adrenergic receptors in the body. These receptors are predominantly found in the lungs, but also in other parts of the body, including the immune system. Clenbuterol’s ability to modulate immune responses and reduce inflammation has sparked interest in its potential use for treating autoimmune disorders like MS. Studies have shown that clenbuterol can improve motor function, increase muscle mass, and reduce fatigue in MS patients, among other benefits. However, the long-term effects of clenbuterol on MS progression and cognitive function are yet to be fully understood.

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The purpose of this study is to assess the safety and tolerability of clenbuterol (taken by mouth) in subjects with ALS (amyotrophic lateral sclerosis) and to assess the effectiveness of clenbuterol with regard to motor function in subjects with ALS. Subjects will be in this study approximately 24 weeks. Multiple sclerosis (MS) is a long-lasting (chronic) disease of the central nervous system. It is thought to be an autoimmune disorder, a condition in which the body attacks itself by mistake. MS is an unpredictable disease that affects people differently. Some people with MS may have only mild symptoms. Multiple sclerosis (MS) is a chronic autoimmune disease of the central nervous system (CNS) characterized by inflammation, demyelination, gliosis, and neuronal loss. [1] Pathologically, perivascular lymphocytic infiltrates, and macrophages produce degradation of myelin sheaths that surround neurons. Epstein–Barr virus as a leading cause of multiple sclerosis: mechanisms and implications. Multiple sclerosis continues to be a challenging and disabling condition but there is now greater understanding of the underlying genetic and environmental factors that drive the condition, including low vitamin D levels, cigarette smoking, and obesity. Amyotrophic Lateral Sclerosis / drug therapy* Clenbuterol / adverse effects Clenbuterol / therapeutic use*. Multiple sclerosis (MS) is a chronic inflammatory disease of the CNS leading to demyelination and axonal degeneration. An increasing body of evidence suggests that lipid metabolism is associated with adverse clinical and MRI outcomes in MS. Whether progression independent of relapse activity (PIRA) heralds earlier onset of secondary progressive multiple sclerosis (SPMS) and more rapid accumulation of disability during SPMS remains to be determined. We investigated the effects of clenbuterol, a β2-adrenoceptor agonist with known anabolic and neuroprotective properties, on G93A-SOD1 mice, a transgenic murine model of familial amyotrophic lateral sclerosis (ALS). We investigated the effects of clenbuterol, a beta2-adrenoceptor agonist with known anabolic and neuroprotective properties, on G93A-SOD1 mice, a transgenic murine model of familial amyotrophic lateral sclerosis (ALS). Relative to saline-treated vehicle controls (0. Article Figures/Media Metrics 91 References 1267 Citing Articles Multiple sclerosis is the most prevalent chronic inflammatory disease of the central nervous system (CNS), affecting more than 2. OBJECTIVE: Clenbuterol, a beta-agonist, has plausible mechanisms for treating amyotrophic lateral sclerosis (ALS). In this highly inclusive open-label trial (NCT04245709), we aimed to study the safety and efficacy of clenbuterol in patients with ALS

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